A study conducted by Zou et al. [37] has suggested that CHM could inhibit the infiltration of lymphocytes and neutrophils in the fallopian tubes of PID-like rats, reduce the release of IL-1β, IL-6, IL-8, and MCP-1, promote the production of LXA4, and found that CHM could regulate LPS-stimulated NF-κB signaling activity and promote FPR2 expression in THP-1 cell line, therefore contributing its anti-inflammatory effect. Here, NFKB1 is linked to pelvic inflammatory disease.