In the DNFB-ACD model, EA at ST36 decreased proinflammatory cell infiltration and proinflammatory cell densities in the dermis and Th1-type cytokines including IFN-γ, TNF-α, IL-1β, and Th2-type cytokines such as IL-4, IL-5, and IL-10 in ear tissues. The gene discussed is NELFCD; the disease is granular corneal dystrophy type II.