In a recent study investigating lncRNAs harboring m6A, LNC942 directly recruited METTL14, a core member of m6A methyltransferase complexes, accompanied by elevated epigenetic m6A methylation modification levels in breast cancer cells, subsequently accelerating cell proliferation and colony formation and decreasing cellular apoptosis rates through LNC942-METTL14-CXCR4/CYP1B1 signaling axis [94]. The gene discussed is METTL14; the disease is breast cancer.