PPP1R14C and neoplasm: Notably, similar to PPP1R14C depletion, silencing PRKCI suppressed cell proliferation, colony formation, anchorage‐independent growth, invasion, migration, and cell‐cycle transition in human and mouse TNBC cells (Figure 6D–H and Figure S4B–E), suggesting that PRKCI, being an upstream activator of PPP1R14C, did play a tumour‐promoting role in TNBC progression.