CD34 and infection: The hypothesis that miR-US5-2-mediated Gab1 down-regulation has a critical role in switching off the HCMV latency program in favor of a lytic infection is also supported by data showing that pharmacological MAPK and PI3K inhibitors enhance HCMV reactivation in CD34+ hematopoietic progenitor cells (Buehler et al., 2019).