DDIT3 and non-small cell lung carcinoma: Licochalcone A induced apoptosis in non-small cell lung cancer (NSCLC) cells by promoting autophagy and simultaneously enhancing the expression of the endoplasmic reticulum stress-related mediator C/EBP homologous protein (CHOP), which is known to clear damaged cells by triggering both apoptotic and autophagic pathways [85].