For example, the favorable group for pulmonary fibrosis includes IL-1β, IL-4, IL-6, IL11, IL-13, IL-17A, IL-15, and IL-33; in contrast, the other group with anti-fibrotic function has IL-7, IL-10, IL-12, and IL-27 (Borthwick, 2016; Steen et al., 2020; She et al., 2021). This evidence concerns the gene IL10 and pulmonary fibrosis.