A growing body of research has identified a number of therapeutic approaches for the treatment of defective bone growth in achondroplasia, the most common form of dwarfism.2 Our present results provide strong evidence for a potential therapeutic effect from (-)-epicatechin on the cartilage growth plate and ciliogenesis in FGFR3-related disorders. This evidence concerns the gene FGFR3 and achondroplasia.