STIM1 and cervical cancer: For example, the knockout of STIM1 inhibits the proliferation (50–60%), colony formation (≈80%), and invasion (≈50%) of hepatocellular cancer SMMC7721 and HepG2 cells.[32] STIM1 knockdown inhibits cell proliferation (≈40%) at day 3 post‐transfection into cervical cancer SiHa cells.[33] In U87 MG cells, we found that knockout of both STIM1 and STIM1β variants (pan‐STIM1‐KO) caused ≈80% reduction in SOCE (Figure S13b,c, Supporting Information), as opposed to ≈20% reduction upon STIM1β depletion.