CCR2 and experimental autoimmune encephalomyelitis: Interestingly, a recent study with a mouse model of experimental autoimmune encephalomyelitis (EAE) revealed that the conditional deletion of Csf2rb in CCR2+Ly6C+ classical monocytes inhibited the neuroinflammation, but the conditional deletion of Csf2rb in other myeloid cells including cDCs and Mo-DCs did not (74).