Ceftriaxone (IV), a cephalosporine antibiotic, that increases the activity of the EAAT2 gene promoter resulting in elevated EAAT2 expression that, in turn, reduces glutamate excitotoxicity,33 significantly improved the ALS phenotype of Tg‐SOD1G93A mice and extended their lifespan by 10 days.34 Here, SLC1A2 is linked to amyotrophic lateral sclerosis.