Our in vivo results showed that the miR‐204–/− mice are susceptible to CH/CD following TAC (Figure 2) and have lower surface availability of APJ despite an increase in the total APJ expression (Figures 4C and 7A), and both phenotypes are reversed by cardioselective overexpression of miR‐204 (Figures 3 and 7C). The gene discussed is APLNR; the disease is cyclic hematopoiesis.