On the other hand, small-cell SNECs and SMARCB1-deficient sinonasal carcinomas seem to be molecularly distinct from IDH2-mutated carcinomas, as supported by the distribution of ARID1A mutations, which were common in small-cell SNEC but not among IDH2-mutant cancers [88]. This evidence concerns the gene IDH2 and nasal cavity and paranasal sinus carcinoma.