Correlative analysis of H2AZ levels with various GBM genotypes revealed a significant, positive association with TP53 mutation and PDGFRA amplifications, but not IDH1 mutation, promoter methylation of MGMT, CDK4/CDK6 amplifications and PTEN mutation in GBM (Supplementary Fig. S1B–G). The gene discussed is TP53; the disease is glioblastoma.