In addition to the previously mentioned results, Faul, C., et al. evaluated the relation between FGF-23 and the pathogenesis of left ventricular hypertrophy (LVH) in humans, in patients from both the Chronic Renal Insufficiency Cohort (CRIC) study—a prospective cohort study conducted with CKD individuals [92]—and studies conducted by their group (Faul C and coworkers) [64]. This evidence concerns the gene FGF23 and chronic kidney disease.