XIST and neoplasm: In ovarian cancer, XIST, miR-140-5p and the transcriptional factor FOXP3 engage a ceRNET, driving ovarian cancer cell progression: XIST was found upregulated in cancer tissues and correlated to poor prognosis of patients; conversely, miR-149-3p was found downregulated with the consequent upregulation of its target, FOXP3; XIST knock-down elevated miR-149-3p to suppress migration and invasion of ovarian cancer cells, and restricted tumor growth in vivo [85].