More than thirty human proteins ranging from globular proteins to unstructured peptide molecules are associated to amyloid disorders (e.g., amyloid β peptide to AD, α-synuclein to Parkinson’s Disease, transthyretin for familial transthyretin amyloidosis), but it is now widely accepted that the ability to form amyloid aggregates is a rather general property of polypeptide chains [5,7]. Here, TTR is linked to Parkinson disease.