Another recent finding [131] also revealed that via modulating the miR-766-5p/CDKN1A axis, METTL3 could modify long non-coding RNAs (lncRNAs) nuclear-enriched transcript 1 (NEAT1), thus controlling the progressive process of CML disorder and playing an oncogenic role in CML. The gene discussed is CDKN1A; the disease is chronic myelogenous leukemia, BCR-ABL1 positive.