TF and Epstein-Barr virus infection: Indeed, it was reported that during an Epstein–Barr virus (EBV) infection on human B-cells, some TF encoded by EBV, as well as some host cell TF activated by this virus, are upregulated, leading to the formation of viral oncogene-mediated SE, particularly near pro-survival and antiapoptotic oncogenes, such as RUNX3, MYC, and BCL2 [34].