In addition, compared with B6 mice, Lm-infected B6.Il18−/−, B6.Nlrp3−/−, and B6.Batf3−/− mice showed reduced levels of serum IFNγ without any effect on the bacterial burden at 24 h post-infection, demonstrating that these factors also regulate IFNγ production during Lm infection. Here, BATF3 is linked to infection.