Differently, the combined treatment of ELP with the synthetic glucocorticoid dexamethasone (DEX) and the glutathione synthesis inhibitor L-buthionine-(S,R)-sulfoximine (BSO) resulted in liver steatosis with increased levels of plasma alanine aminotransferase (ALT), accompanied by myeloperoxidase-positive cells infiltrating hepatic tissue and increased oxidative stress-related factors, such as hepatic heme oxygenase-1, serum hydrogen peroxide and hepatic malondialdehyde [65]. Here, GPT is linked to fatty liver disease.