Molecules that could be used as NF-κB-inhibiting tool compounds for further investigation should possess an inhibitory potency comparable to that of parthenolide [17,18,19,20], a sesquiterpene lactone from feverfew Tanacetum parthenium, which modulates the NF-κB-mediated inflammatory response, e.g., in experimental atherosclerosis [21], and induces apoptosis in acute myelogenous leukemia (AML) cells [22,23]. This evidence concerns the gene NFKB1 and acute myeloid leukemia.