The data reveal that CML-HSA induced the expression of the inflammatory genes TNFα, IL-1β, IL-6, and iNOS by 4-, 30-, 3-, and 4-fold, respectively, in the vehicle control compared to the untreated group; both concentrations of astaxanthin inhibited CML-HSA-induced inflammatory gene expression two-fold or more compared to the vehicle control (Figure 2C–F). This evidence concerns the gene TNF and chronic myelogenous leukemia, BCR-ABL1 positive.