Because of its antioxidative activity, GA reduced oxidative stress, inhibited fatty acid synthesis, increased the expression of NFE2L2, and alleviated steatosis, thus exerting protective effects against dysfunctional lipid metabolism by directly targeting NFE2L2, which could be partially attributable to the downregulation of miR-34a-5p expression in the liver. This evidence concerns the gene NFE2L2 and steatosis.