NFE2L2 and intracerebral hemorrhage: Regarding the role of NRF2 in pathological states, knockout mice showed lower survival rates, more severe injury, and increased inflammation after intracerebral hemorrhage [58,59], neonatal hypoxia–ischemia [60], traumatic brain injury [61], and middle cerebral artery occlusion [62], highlighting the critical role of NRF2 in maintaining neuronal survival after oxidative insults.