Li and Wang (2021) reported GABPB1-AS1 competitively bound to miR-330 and reinforced the ZNF367 expression, thereby facilitating glioma cells progression. In cervical cancer, E6-induced GABPB1-AS1 overexpression facilitated tumor proliferation and invasion (Ou et al., 2020). However, the function and mechanism of NORAD and GABPB1-AS1 in thyroid cancer have not been reported, and its role in PTC needs further exploration. Here, GABPB1 is linked to glioma.