For instance, S100A4/non-muscle myosin II signaling has been associated with epithelial–mesenchymal transition and stemness in uterine carcinosarcoma [18], while the inhibition of S100A8 expression is reported to promote apoptosis through the suppression of AKT phosphorylation [19]. The gene discussed is AKT1; the disease is uterine carcinosarcoma.