BECN1 and myocardial infarction: Mitochondria from Fundc1 KO mouse hearts after acute MI showed significantly decreased Δψm and increased ROS production compared with mitochondria from either Beclin1+/− hearts or wild‐type hearts after acute MI (Figure 4B,C), suggesting that Fundc1 deficiency caused mitochondrial impairment exacerbates mitochondrial dysfunction after acute MI.