We calculated a Pol II pausing index for CRTC2‐target genes in normal kidney and ADPKD kidney by re‐analyzing the Pol II ChIP‐seq data from our earlier study.[33] The pausing index was determined by comparing Pol II enrichment on gene promoter‐proximal regions with that on the corresponding gene bodies. Here, CRTC2 is linked to autosomal dominant polycystic kidney disease.