PSMB9 and breast cancer: As a result, seven candidate genes (JUN, FOS, FOSB, EGR1, ZFP36, CFD, and PPARG) were significantly upregulated in luminal A type of BC and normal breast‐like samples compared with luminal B, HER2+, and TNBC, while CD27, PSMB9, and SELL were opposite (Figure 7), as well as in TP53‐normal relative to TP53‐mutant group (Figure S3).