For instance, Xie et al.67reported that JUN was upregulated in TNBC than non‐TNBC tumors, and the JUN N‐terminal kinase (JNK)/JUN signaling pathway might regulate cancer stem‐like cell phenotype and tumorigenesis through Notch1 signaling pathway in TNBC, while Anders et al.68demonstrated that hyper‐phosphorylation of JUN by JNK inhibited tumor cell migration and invasion. This evidence concerns the gene JUN and neoplasm.