It is well reported that Myf‐5 is required during the regenerative process to allow for the proliferation of myogenic populations (Pax7+, which was also shown to be elevated in CKD derived cells) in order to facilitate muscle regeneration43 and that this process can be driven by inflammatory stimuli, such as TNF‐α and IL‐6.43 This evidence concerns the gene MYF5 and chronic kidney disease.