TMEM120A and obesity due to melanocortin 4 receptor deficiency: For example, genes at DRs normally moving to the NE to become repressed that failed to move and failed to be repressed in the Tmem120a knockdown included Cpe that yields obesity in mice when defective48 and Dctd that has altered genome interactions in fat morbidities49 and increased SNPs in childhood obesity50 (Fig. 5d).