A KEGG enrichment analysis of these monocyte data confirmed the depletion of IFN-related pathways (influenza, measles, cytosolic DNA sensing pathway) by DI treatment, but also of other pro-inflammatory pathways (TLR signaling, cytokine-cytokine receptor interaction, NF-kB signaling) in infected and uninfected cells (S23 Fig). Of note, DI treatment apparently stimulated ribosome function in both infected and control monocytes. Here, NFKB1 is linked to influenza.