Re-analysis of a published dataset from a model using a different IAV strain (H3N2) and histologically normal lung tissue [38] revealed a pronounced induction of ACOD1 and TNFAIP3 expression, but (as opposed to in vivo infection of mouse lung) not induction of HMOX1 or 2 (S16 Fig). This evidence concerns the gene TNFAIP3 and infection.