For example, mutation of the golgin GMAP210 (TRIP11) gives rise to two skeletal disorders in humans, namely achondrogenesis 1A (ACG1A) and odontochondrodysplasia (ODCD), which are both associated with defective trafficking and glycosylation of matrix proteins (Smits et al., 2010; Wehrle et al., 2019). This evidence concerns the gene TRIP11 and achondrogenesis type IA.