This finding also fits with RASopathy-specific models, such as the SOCIAL model originally developed by Beauchamp and Anderson [105] and adapted by Chisholm et al. [53] as a relevant model for NF1; these models account for the internal, external, and neurobiological factors that influence cognitive functions and how those interconnected cognitive functions (e.g., attention/executive functions, communication, socio-emotional factors) contribute to the social deficits observed in individuals with RASopathies. Here, NF1 is linked to RASopathy.