Although recently suggested to be specific to fibrotic human livers (Ramachandran et al., 2019), we identified LAMs in all patients profiled with scRNA-seq, but there was a trend toward increased proportions of LAMs in the livers with >10% steatosis (Figure S8D) consistent with the increased population of LAMs in murine NAFLD (Figures S6J–S6N). Here, LAMB2 is linked to metabolic dysfunction-associated steatotic liver disease.