NFKB1 and myotonic dystrophy type 1: In our model, we found that IL6-JAK-STAT3 signalling and TNFA signalling via NFKB were positively enriched in both experimental groups, albeit that the IL6-JAK-STAT3 signalling may have been more strongly upregulated in the control group, while the interferon alpha response was exclusively enriched in the DM1 cells.