In our model, we found that IL6-JAK-STAT3 signalling and TNFA signalling via NFKB were positively enriched in both experimental groups, albeit that the IL6-JAK-STAT3 signalling may have been more strongly upregulated in the control group, while the interferon alpha response was exclusively enriched in the DM1 cells. Here, TNF is linked to myotonic dystrophy type 1.