Among all patients, 20.2% had thyrotoxicosis and 5.2% had hypothyroidism, and they found that thyrotoxicosis was associated with rising interleukin-6 levels, which indicates that COVID-19 may be associated with a high risk of thyrotoxicosis related to systemic immune activation including IL-1, natural killer (NK) cells [7, 8], IL-6, T-cell-mediated immune responses, and proinflammatory cytokines [9–11], while the postviral inflammatory reaction may be an inducing factor of thyroid dysfunction. This evidence concerns the gene IL6 and COVID-19.