GAD1 and Dravet syndrome: To assess if this defect characterizes also our reversible mouse model and drives the characteristic associated phenotype, we performed whole-cell patch clamp analysis on acute brain slices of Scn1aStop/+ (129 Sv) mice crossed with Gad67-GFP knock-in mice22 (C57Bl6/N) first at P18-P23, when DS symptom start to develop.