Interestingly, the lung homogenates of IPF patients exhibited 6.4-fold and 10.8-fold higher ACP5a and ACP5b expression than those of control subjects, respectively and markedly higher expression levels of COL1A1 and α-SMA, markers of fibrosis, were also noted in IPF patients (Fig. 1c). The gene discussed is ACTA1; the disease is idiopathic pulmonary fibrosis.