ACKR5 and neoplasm: As CXCL9 and CXCL10 are the only functional ligands for CXCR3 in C57BL/6 mice, complete reversal of the anti-tumor phenotype and improved T cell infiltration in GPR182-deficient mice with CXCR3 blockade demonstrates that alteration in the abundance of these two chemokines in the TME is driving the observed phenotype.