The collective data offers a new insight into the anti-tumor role ofmiR-7-5p, and further reveals that exosomal miR-7-5p secreted by BMSCs suppresses theproliferative activity and promotes the apoptosis of AML by targeting OSBPL11 to inhibitPI3K/AKT/mTOR signaling pathway phosphorylation. The gene discussed is AKT1; the disease is acute myeloid leukemia.