Thus, allergic asthma is typically related to Th17 cells expressing several effector cytokines such as IL-17A and 17F, IL-22, IL-26, and granulocyte-macrophage colony-stimulating factor (GM-CSF) [13]; conversely, atopic dermatitis is clearly a Th2-type-driven inflammation, while allergic rhinitis is classically defined by the imbalance between Th1/Th2 response, with a predominance of Th2 and Th17 cells [14,15,16]. This evidence concerns the gene IL17A and atopic eczema.