At the same time, the results of experimental studies show that proliferatively active BrdU, H3Ser10, and aurora B kinase-labeled CMCs are registered in the heart of animals kept under hypoxic conditions, and the scar size after myocardial infarction induced by ligation of the left coronary artery is significantly less than in animals under conditions of normoxia [137]. The gene discussed is AURKB; the disease is myocardial infarction.