In addition, FLU suppressed the cell migration and reversed the epithelial-mesenchymal transition (EMT) phenotype by decreasing the expression of mesenchymal markers (β-catenin, N-cadherin, and Vimentin) and promoting the expression of the epithelial and differentiation marker (Keratin 18) in CS-like cells-enriched breast cancer, indicating that FLU inhibited breast cancer metastasis. The gene discussed is VIM; the disease is breast carcinoma.