Neutrophils are recruited to the site of the tumor bed by the interplay of different chemokines that are responsible for their maturation, migration, proliferation, and function, including CXCR2 (interleukin 8 receptor, beta) granulocyte-colony stimulating factor (G-CSF) and interleukin 17A (IL-17A) [5,6,7]. This evidence concerns the gene CXCR2 and neoplasm.