On the other hand, single bolus administration of BMDSCs one week after induction of glomerulonephritis with anti-Thy1 injection failed to reduce oxidative stress, apoptosis and markers of glomerular injury or to restore glomerular nephrin expression even though BMDSC administration reduced the expression of renal T cells, IL-17 and activated macrophages. This evidence concerns the gene IL17A and glomerulonephritis.