Furthermore, MSCs had striking effects on glomerular pathology, as half of the glomeruli were spared from any lesions [31,32]; in contrast, in non-MSC-treated rats, anti-Thy1 induced mesangioproliferative glomerulonephritis that is characterized by an initial mesangiolysis followed by reparative overgrowth of mesangial cells [31,32,33]. Here, THY1 is linked to glomerulonephritis.