Compared to that of CD3− MDMs, H37Ra (MOI 1) increased the frequency of CD3+TCRαβ−CCR2+ MDMs, as well as their ligand, CCL2 (Figure 5A,B, left, respectively), whereas H37Rv infection (at both MOIs) induced an increase in the frequency of CD3+TCRαβ+CCR2+ MDMs compared with that of CD3− MDMs (asterisk), and the CCL2 levels were similar (Figure 5A,B, right, respectively), although they were lower than those with H37Ra infection. The gene discussed is CCR2; the disease is infection.