The function of some genes upregulated in the Catulin-GFP reporter cells in cancer progression is still controversial such as for Cav1, which can act as both tumor suppressor, by regulating integrin β1- and Src-mediated cell–cell and cell–matrix interactions [67,68,69] and tumor-promoting molecules by affecting miR-133a [70,71]. The gene discussed is CAV1; the disease is neoplasm.