IL6R and renal fibrosis: The most important findings include members of the miR-30 family, followed by miRNAs related to processes and cytokines previously deemed important in AAV pathogenesis, namely monocyte/macrophage polarization (hsa-miR-142-5p, hsa-miR-150-5p, hsa-miR-181a-5p, hsa-let-7a-5p), T-cell activation and differentiation (hsa-miR-542-5p) and related interleukin 6 receptor (IL-6R) signaling (hsa-miR-204-5p), the B-cell activating factor (BAFF, hsa-miR-30a-3p), renal fibrosis (hsa-miR-21-3p and hsa-miR-150-5p) and endothelial injury (hsa-miR-204-5p).